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. 2020 Nov 23;10:20386. doi: 10.1038/s41598-020-77411-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Circulating triglyceride and retinyl ester metabolism in wild type and Cd36^−/− mice. Postprandial (A) plasma triglyceride and (B) plasma retinyl ester clearance was determined in saline and p-407 injected mice. (C) Retinyl esters were also normalized to plasma TG (WT Saline, n = 7; Cd36^−/− Saline, n = 6; WT p-407, n = 7; Cd36^−/− p-407, n = 5 mice per group). Hepatic VLDL D) triglyceride secretion and (E) retinyl ester secretion were determined in p-407 injected mice. (F) retinyl esters were normalized to triglyceride (n = 6 mice per group). Data is shown as mean ± S.D. and significance determined by two-way ANOVA (A–C; *p < 0.05 denotes a significant post-test result between genotypes within the same treatment) or Student’s t-test (D–F; ***p < 0.001).