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. 2020 Nov 23;10:20382. doi: 10.1038/s41598-020-77290-w

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Photobiomodulation protects against mitochondrial dysfunction and oxidative stress in the P23H retina. (a) shows a representative max projected NADH (upper panel), FAD (middle panel), and redox ratio (NADH/FAD) images (lower panel) for normal (SD), P23H transgenic, and 830 nm PBM-treated transgenic retinas. (b) shows the corresponding histograms for retinas, comparing normal (SD), P23H transgenic, and 830 nm treated transgenic eyes. (c) shows a bar graph plot comparing the mean values of the histograms of max projected images from SD, P23H transgenic, and 830 nm PBM-treated P23H transgenic rat eyes. The results document a significant decrease in the ratio between normal (non-dystrophic SD eye) and diseased eyes (**p < 0.01), which is restored to control values by 830 nm treatment (*p < 0.01). Error bars: SE.