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. 2020 Nov 24;4(22):5877–5887. doi: 10.1182/bloodadvances.2020002646

Table 1.

A summary and comparison of proposed immunomodulatory mechanisms of MSCs in studies reviewed herein

Cell/mechanism Immune effect Proposed MOA Model Reference
Adaptive immunity
 CD4+/CD8+ suppression ↓T-cell proliferation, ↑survival IFN-γ priming/IDO metabolism In vitro/vivo 22,23
 CD4+/CD8+ death ↑T-cell apoptosis? IFN-γ priming/IDO metabolism In vitro 47
 T cell–Treg polarization ↓Inflammation? IFN-γ/cytokine/paracrine In vitro 48
 B-cell suppression ↓B proliferation IFN-γ/cytokine/paracrine In vitro 43,44
Innate immunity
 Polarize M2 macrophages ↓Inflammation Monocyte/IFN-γ MSC priming In vitro 19,21,53
 Macrophage recruiting ↑Tissue repair Chemotaxis to damaged tissue In vitro/vivo 19,24,54
 Monocyte recruiting ↑Tissue repair Chemotaxis to damaged tissue In vitro/vivo 19,24,54
 Apoptotic MSCs Immunosuppression Phagocytic-induced suppression In vitro/vivo 55-57

IDO, indoleamine 2,3-dioxygenase; MOA, mechanism of action; Treg, regulatory T cell; ↑, increased; ↓, decreased.