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. 2020 Nov 12;14:580206. doi: 10.3389/fncel.2020.580206

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Copyright © 2020 Liu, Huang, He, Zhuo, Chen, Ge, Duan, Lu and Hu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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OM-MSCs attenuated OGD/R-induced ROS overproduction and mitochondrial dysfunction in N₂a cells. (A–B) Intracellular ROS levels in N₂a were measured by staining with DCFH-DA, and the determination of ROS levels was carried out using a flow cytometer. (C–E) The total SOD, GSH-PX, and MDA activities in N₂a were assessed. (F) The ATP content in N₂a cells was assessed using an ATP assay kit, and data are expressed as a percentage of the control. (G,H) N₂a was incubated with JC-1, and the determination of mitochondrial membrane potential was carried out using a flow cytometer. The mitochondrial membrane potential in each group was calculated as the ratio of red to green fluorescence. All data are displayed as mean ± SEM (n = 3). (^∗∗P < 0.01 vs. control; ^##P < 0.01 vs. OGD/R).