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. 2020 Nov 26;20:569. doi: 10.1186/s12935-020-01620-1

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — LINC00152 mediates the EZH2/ZEB1 axis to promote EMT and EC cell resistance to L-OHP. a tumor growth rate curves of nude mice xenografted with EC cells (n = 6). b the mRNA expression of LINC00152, EZH2, and ZEB1 in tumor tissues of nude mice xenografted with EC cells (n = 6) detected by RT-qPCR. c the protein expression of EZH2 and ZEB1 in tumor tissues of nude mice xenografted with EC cells (n = 6) detected by Western blot analysis. * p < 0.05 vs. The oe-NC + si-NC group; # p < 0.05 vs. the oe-NC + si-NC + L-OHP group; & p < 0.05 vs. the oe-LINC00152 + si-NC + L-OHP group. Data are presented as mean ± standard deviation of three technical replicates