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Proposed mechanism of TN-C under physiological and pathological conditions. EGFR, epidermal growth factor receptor; TLR4, Toll-like receptor 4; PDGF, platelet-derived growth factor; PDGFR, platelet-derived growth factor receptor; ECM, extracellular matrix; FAK, focal adhesion kinase; PKB, protein kinase B; PKC, protein kinase C; MAPK, mitogen-activated protein kinase.
