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. 2020 Sep 12;252(3):e5529. doi: 10.1002/path.5529

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© 2020 The Authors. The Journal of Pathology published by John Wiley & Sons, Ltd. on behalf of The Pathological Society of Great Britain and Ireland.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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Mutation and CNV profiles in non‐ampullary duodenal adenomas and intramucosal adenocarcinomas. Upper panels show histological types, Vienna classification, and CIMP status. Middle panels show summarized results for targeted sequencing of cancer‐related genes in tumors with the indicated histological types. Lower panels show frequently detected CNVs. Frequencies of CIMP status as well as mutations and CNVs in respective genes are shown on the right. CIMP, CpG island methylator phenotype; GCA, gastric‐type intramucosal adenocarcinoma; PGA, pyloric gland adenoma; SCA, small intestinal‐type intramucosal adenocarcinoma; SHA, small intestinal‐type high‐grade adenoma; SLA, small intestinal‐type low‐grade adenoma.