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. 2020 Nov 27;20:1160. doi: 10.1186/s12885-020-07590-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 9 — a. Expression of IGF2BP2 and IGF2BP3 in pancreatic cancer and normal cell lines. b. Expression of IGF2BP2 and IGF2BP3 in pancreatic cancer and normal cell lines assessed by western blot analysis. c. Proliferation of SW1990 and ASPC-1 cells detected by CCK-8 assays. d. Proliferation of SW1990 (NC vs SiIGF2BP2) (NC vs SiIGF2BP3) cells detected by colony formation assays. d. Invasion of SW1990 (NC vs SiIGF2BP2) (NC vs SiIGF2BP3) cells detected by transwell assays (magnification 400x). Data are presented as the mean ± SD of at least three independent measurements. *P < 0.05, **P < 0.01 and ***P < 0.001. P < 0.05 was considered statistically significant