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. 2020 Nov 27;11:6083. doi: 10.1038/s41467-020-19855-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Clinicopathologic characteristics of the included 53 T1 stage LUAD patients. b Micro-dissection was performed to separate pre-invasive and invasive components within MPNs. c, d Number and type of somatic mutations detected in 6 whole MPNs and 35 micro-dissected MPN components of the cases using 1021-panel (Phase 1; c), and somatic mutations detected in 2 whole MPNs and 78 micro-dissected MPN components of the cases using 425-panel (Phase 2; d). Inv Invasive, Pre-Inv pre-invasive, AAH atypical adenomatous hyperplasia, AIS adenocarcinoma in situ, MIA minimally invasive adenocarcinoma, IAC invasive adenocarcinoma.