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AITC pretreatment reduces APAP-induced phosphorylation of c-Jun N-terminal kinase (JNK) and tumor necrosis factor-α (TNF-α) expression. Immunoblotting was performed to evaluate the phosphorylation of JNK as an APAP-induced damage marker in liver tissues from (A) 13 h APAP treatment set. (B) TNF-α expression level was analyzed by RT-PCR in 13 h APAP treatment set.
