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. 2020 Nov 27;477(22):4425–4441. doi: 10.1042/BCJ20200656

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© 2020 The Author(s)

This is an open access article published by Portland Press Limited on behalf of the Biochemical Society and distributed under the Creative Commons Attribution License 4.0 (CC BY). Open access for this article was enabled by the participation of University of Edinburgh in an all-inclusive Read & Publish pilot with Portland Press and the Biochemical Society under a transformative agreement with JISC.

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Figure 3. — Levels of ATP inhibition differ between PFK-M (panel A), PFK-L (panel B), and PFK-P (panel C), at ATP concentrations of 0.5 mM (blue squares with solid lines), 1 mM (red circles with dotted lines), and 2 mM (green triangles with dot-dash lines). (n = 3). Note that y-axis scales differ between Panels.

Inline graphic — Levels of ATP inhibition differ between PFK-M (panel A), PFK-L (panel B), and PFK-P (panel C), at ATP concentrations of 0.5 mM (blue squares with solid lines), 1 mM (red circles with dotted lines), and 2 mM (green triangles with dot-dash lines). (n = 3). Note that y-axis scales differ between Panels.