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. 2020 Nov 5;23(1):31. doi: 10.3892/mmr.2020.11669

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — SNHG20/miR-495 axis regulates CRC progression. (A) MTS and (B) colony formation assays indicated that SNHG20 knockdown decreased the cell viability of HCT116 and SW480 cells, which could be reversed by treatment of the cells with the miR-495 inhibitor. (C) Transwell assays showed that SNHG20 knockdown inhibited cell invasion and migration of HCT116 and SW480 cells. This effect could be abolished following treatment of the cells with the miR-495 inhibitor. *P<0.05 and ***P<0.001. SNHG20, small nucleolar RNA host gene 20; CRC, colorectal cancer; miR, microRNA; si-, small interfering RNA; NC, negative control.