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. 2020 Dec 2;13:53. doi: 10.1186/s13072-020-00375-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Profiles of sperm DNA methylomes from the control and arsenic F1 mice. a Cluster analysis of methylomes of F1 sperm and normal and tumor tissues from F2 livers. b Average methylation levels of all CpGs in each chromosome in the F1 sperm. The differences in the methylation levels between the control and arsenic groups were tested by Student t test (*p < 0.05). The number (c) and % of hypo/hyperDMCs in all CpGs (d) in each chromosome in the F1 sperm