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. 2020 Dec 3;17:369. doi: 10.1186/s12974-020-02043-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — TMEV-injected animals exhibit scarring and reactive NG2 cells and microglia/macrophages in the hippocampus at 14 dpi. a NG2 cells (green) and microglia/macrophages (red) in the CA1 region of the hippocampus of PBS-injected (top) and TMEV-injected (bottom) mice at 14 dpi. b TMEV-injected mice showed significantly greater immunoreactivity for NG2 compared to PBS-injected mice. c TMEV-injected mice showed significantly greater immunoreactivity for IBA1 compared to PBS-injected mice. Images are ×20 maximum-intensity projections of 12 μm confocal z stacks. Scale bars = 100 μm