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. 2020 Nov 20;23(1):73. doi: 10.3892/mmr.2020.11711

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Copyright: © Ye et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Effect of the mitophagy inhibitor Mdivi-1 on cell death and mitochondrial function in OGD/R-injured primary cortical neurons. (A) Quantification of fluorescence intensity of mitochondrial superoxide. (B) TMRE fluorescence intensity indicated mitochondrial membrane potential. (C) Viability and (D) apoptosis of neurons subjected to OGD/R simulation in the presence or absence of Mdivi-1. Data are presented as the mean ± SEM (n=3). *P<0.05 vs. the control. ^#P<0.05 vs. OGD/R. ^†P<0.05 vs. OGD/R + resveratrol. OGD/R, oxygen/glucose deprivation/reoxygenation; Mdivi-1, mitochondrial division inhibitor-1; TMRE, tetramethylrhodamine ethyl ester.