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. 2020 Nov 20;23(1):74. doi: 10.3892/mmr.2020.11712

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — PFKP promotes migration, invasion and EMT of non-small cell lung cancer cells. (A) Wound healing assay was used to detect the migration of H23 and H1299 cells. (B) Transwell assay was used to detect the invasion of H23 and H1299 cells. (C) Expression levels of EMT-related proteins (N-cadherin, vimentin and E-cadherin) in H1299 and H23 cells were determined by western blot analysis. Data are presented as the mean ± SD from three independent experiments. **P<0.01 vs. control group; ^##P<0.01 vs. si-NC group; ^$$P<0.01 vs. pCMV6-ENTRY group. PFKP, platelet isoform of phosphofructokinase; EMT, epithelial-mesenchymal transition; si, small interfering RNA; NC, negative control.