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Copy number variations, especially the heterozygous deletion, contributes to the downregulation of dexamethasone responsive genes. (A) Heterozygous and (B) homozygous amplification and deletion of dexamethasone responsive genes in cancers; (C) copy number variation is tightly correlated with gene expression of dexamethasone responsive genes. Only events with over 5% heterozygous amplification or deletion were included for analysis.
