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. 2020 Nov;8(21):1365. doi: 10.21037/atm-20-3848

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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The tumorigenic capability of Huh7 cells was inhibited by LINC00844 overexpression (A), which was indicated by the tumor volume (B) and tumor weight (C). Positive rates of Ki-67 (D) and cyclin D1 (E) immunohistochemistry stain were higher in the LINC00844 knockdown group than in the LINC00844 upregulation group. *, P<0.05. LINC00844, long non-coding RNA00844.