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. 2020 Nov;8(21):1365. doi: 10.21037/atm-20-3848

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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LINC00844 overexpression promotes the cell cycle arrest of HCC cells (A), and this effect is reversed by LINC00844 knockdown (B). Huh7 (C) and LM3 cells (D) with high LINC00844 expression were transfected with siRNA AZGP1. SiAZGP1 eliminated the inhibitory effect of LINC00844 on cell cycle in Huh7 (E) and LM3 cells (F). *, P<0.05; **, P<0.01. LINC00844, long non-coding RNA00844; AZGP1, zinc-alpha-2-glycoprotein; HCC, hepatocellular carcinoma.