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. 2020 Nov;8(21):1422. doi: 10.21037/atm-20-2509

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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miR-137 contributed to HCC progression. (A) RT-qPCR confirmed the upregulated miR-137 expression in HCC cell lines and the miR-137 overexpression in the HepG2 and HCCLM3 cells transfected with miR-137 mimic compared to miR-NC. ***, P<0.001 vs. miR-NC. MTT (B), Transwell with 0.5% crystal violet staining ×20 (C), and wound healing assay ×4 (D) were conducted to examine the cell proliferation and migration of HepG2 (C) and HCCLM3 (D) cells. **, P<0.01; ***, P<0.001 vs. miR-NC. (E) Survival curves of miR-137. (F) Enrichment analysis of miR-137 predicted target genes. RT-qPCR, reverse transcription-quantitative polymerase chain reaction; miR, microRNA; miR-NC, negative control scramble miR. HCC, hepatocellular carcinoma.