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. 2020 Dec 9;26:123. doi: 10.1186/s10020-020-00245-4

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Representative in situ hybridization of Nhlrc2 in the brain of a 32-week-old male C57BL/6NCrl mouse. a Overview image of ISH with Nhlrc2 probe (red). Red boxes mark the magnified areas: a CA1 layer of hippocampus, b dentate gyrus of hippocampus, c piriform cortex, d cerebellar hemisphere. b Nhlrc2 expression in areas a–d. A strong signal was observed from the pyramidal cell layer of the hippocampus and piriform cortex as well as dentate and cerebellar granule cells. c Negative control from areas a–d