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Possible mechanisms of hydrogen sulfide (H2S) in the healing of diabetic skin wounds. Increasing endogenous H2S production or exogenous H2S supplementation attenuates oxidative stress and inhibits extracellular regulated protein kinases (ERK) 1/2 and p38 phosphorylation to accelerate diabetic skin wound healing. H2S also improves angiogenesis by restoring endothelial progenitor cell (EPC) function to promote skin wound healing.
