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. 2020 Nov 28;21(23):9046. doi: 10.3390/ijms21239046

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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FAHFAs decreased mitochondrial respiratory chain activity in proliferating C2C12 myoblasts. (A) FAHFAs decreased mitochondrial complex IV activity in proliferating C2C12 myoblasts. Enzymatic activity was measured 24 h after FAHFA treatment at the indicated concentration (n = 6 for each group). (B) FAHFAs had no influence on citrate synthase activity in proliferating C2C12 myoblasts. Enzymatic activity was measured 24 h after FAHFA treatment at the indicated concentration (n = 6 for each group). (C) FAHFAs had no influence on ROS production in proliferating C2C12 myoblasts. ROS accumulation was measured using the H2DCF-DA probe 24 h after FAHFA treatment at the indicated concentration (n = 6 for each group). The effect on the groups, including the control group, was tested by one-way ANOVA, followed up by a Fisher’s least significant difference (LSD) test; all the groups, except the control group, were also tested for the effects of dose, FAHFAs, and their interaction by two-way ANOVA test. Results are expressed as means ± SEM.