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. 2020 Dec 11;10:144. doi: 10.1186/s13578-020-00506-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — ATP13A2 increases the invasive capacity of colon cancer cells in vitro. a Representative images (Left panel) and number of invasive cells (Right panel) of HCT-116 cell treated with siNC or siATP13A2. b Representative images (Left panel) and number of invasive cells (Right panel) in cells that overexpress ATP13A2 as and the control cells (ctrl). c The relative expression of ATP13A2, MMP2, MMP9, E-cadherin, Vimentin in mock- and OEATP13A2; siNC and siATP13A2- HCT-116 cell. d The relative protein of MMP2 in mock- and OEATP13A2; siNC and siATP13A2- HCT-116 cell. *, P < 0.05; ***, P < 0.001. ns, no significant