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. 2020 Dec 3;132(23):742–761. doi: 10.1007/s00508-020-01781-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Magnetic resonance tomography in a patient with cardiac transthyretin amyloidosis. Top row: short axis view. Bottom row: four-chamber view. a Functional cine imaging showing pronounced left ventricular hypertrophy. b T1 mapping showing globally increased T1 relaxation times of 1180 ± 38 ms. c Biventricular late gadolinium enhancement with subendocardial enhancement showing characteristic, global patchiness mostly in the basal segments of the myocardium. d Extracellular volume (ECV): globally increased ECV of 58.4%, estimated according to the formula: ECV = (1 − hematocrit) × [1/post-contrast T1 myocardium − 1/native T1 myocardium]/[1/post-contrast T1 blood − 1/native T1 blood]