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. 2020 Dec 11;11(12):1049. doi: 10.1038/s41419-020-03244-9

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A The mRNA level of NCAPH in cervical cancer (CC) (n = 22) and normal cervix (NC) (n = 10) analyzed by real-time PCR. Data are presented as mean ± SD (n = 3). B, C) Immunohistochemical staining of NCAPH in normal cervix (NC) (n = 82), high-grade squamous intraepithelial lesion (HSIL) (n = 34) and invasive cervical cancer (ICC) (n = 165). Scale bars, 100 μm. D ROC curves showed the clear separation between normal cervix and cervical cancer tissues, with an area of 0.8826 (p = 0.0006). E NCAPH expression in several solid tumors analyzed by GEPIA data base. Normal, normal tissues; ESCA, esophageal carcinoma; BRCA, breast invasive adenocarcinoma; HNSC, head and neck squamous cell carcinoma and BLCA, bladder urothelial carcinoma. F Compared with LNM-negative cervical cancer (n = 104), NCAPH displayed higher expression rate in LNM-positive tissues (n = 61) (p = 0.0041). Kaplan–Meier curves for survival of 100 patients with cervical carcinoma. Culminative disease-free survival (G) and overall survival (H).