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. 2020 Dec 12;11(12):1063. doi: 10.1038/s41419-020-03236-9

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a, b. Nuclear separation followed by RT-qPCR and AGE, as well as FISH assay, monitored the subcellular location of LINC01116 in BCa cells. c, d GEPIA 2 analyzed the relationship between two protein-coding genes (ELK3 and HOXD8) and LINC01116 in BLCA tumors. e, f RT-qPCR and western blot analyzed the expression of ELK3 in BCa cells under LINC01116 inhibition. g, h RT-qPCR and western blot analyzed the expression of HOXD8 in BCa cells. i, j Dual-luciferase reporter assay further validated the combination between ELK3/HOXD8 and LINC01116. *P < 0.05, **P < 0.01.