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. 2020 Dec 11;18(4):1559325820972205. doi: 10.1177/1559325820972205

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 6. — AlCl3 treatment significantly enhanced the protein expressions of APP, Aβ1-42, β and γ-secretases and favors amyloid biosynthesis. Coadministration of HGN50 (AlCl3+50 mg/kg HGN) attenuated the AlCl3 mediated amyloid biosynthesis. (a) Representative Western blots, (b) Corresponding graph of relative Western blot intensity in the hippocampus and (c) Corresponding graph of relative Western blot intensity in the cortex. Data are expressed as mean ± SEM (1-way ANOVA followed by Duncan’s multiple range test) for 3 rats in each group. a—p < 0.05 compared to the control, b—p < 0.01 compared to the control, c—p < 0.05 compared to the AlCl3 treated rats, d—p < 0.01 compared to the AlCl3 treated rats.