Skip to main content
. 2020 Nov 28;2020:4640605. doi: 10.1155/2020/4640605

Figure 5.

Figure 5

Recombinant rat ANXA5 reversed the testicular oxidative stress promoting injury of miR-506-3p in DBP-treated rats: (a) H&E staining of testis tissues in DBP-treated rats following Agomir-506-3p and cotransfection of recombinant rat ANXA5 and Agomir-506-3p; (b) DHE staining of testis tissues in DBP-treated rats following Agomir-506-3p and cotransfection of recombinant rat ANXA5 and Agomir-506-3p, ROS exhibit red fluorescence under fluorescent microscope; (c) immunohistochemical staining showed ANXA5 expression level of testis tissues in DBP-treated rats following Agomir-506-3p and cotransfection of recombinant rat ANXA5 and Agomir-506-3p; (d) Western blotting showed the expression levels of Nrf2, ANXA5, HO-1, NQO1, and GST proteins of testis tissues in DBP-treated rats following Agomir-506-3p and cotransfection of recombinant rat ANXA5 and Agomir-506-3p. The data are showed as mean ± SD. ∗Significantly different from the DBP + Agomir-506-3p group. ∗P < 0.05, ∗∗P < 0.01.