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. 2020 Dec 14;39:286. doi: 10.1186/s13046-020-01802-9

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — MIEF2 knockdown suppressed migration and invasion of OC cells. a and b Scratch wound healing and transwell matrigel invasion assays were applied in OVCAR3 and ES2 cells with or without MIEF2 knockdown. siMIEF2, siRNA against MIEF2; siCtrl, control siRNA. c and ds qRT- PCR and western blot analysis for expressions of epithelial markers of E-cadherin and ZO-1, and mesenchymal markers of N-cadherin and Vimentin in OVCAR3 and ES2 cells with or without MIEF2 knockdown. *P < 0.05