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. 2020 Oct 22;2(1):vdaa142. doi: 10.1093/noajnl/vdaa142

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© The Author(s) 2020. Published by Oxford University Press, the Society for Neuro-Oncology and the European Association of Neuro-Oncology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 2. — MRI radiologic features of H3 K27M-mutant DMGs in adults at the time of initial diagnosis. (A) Description of gadolinium enhancement on MRI of the 60 patients. (B) Post-gadolinium (B1) and FLAIR (B2) images from a non-enhancing tumor. A blood vessel courses through the left thalamic tumor (white arrowhead), and periventricular FLAIR hyperintensity indicates transependymal flow secondary to acute obstructive hydrocephalus (red arrowhead). (C) Several examples of DMGs with H3 K27M mutation with different enhancement patterns: nodular (C1), thin rim (C2), heterogeneous (C3), thick rim (C4), diffuse/solid enhancement (C5), and disseminated disease with a conus lesion and leptomeningeal spread (C6).