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. Author manuscript; available in PMC: 2021 Dec 1.
Published in final edited form as: Liver Res. 2020 Oct 17;4(4):191–198. doi: 10.1016/j.livres.2020.10.001

Fig. 3. Antibiotics lowered the abundance of Clostridium which leads to increased ratio of the primary to the secondary bile acids.

Fig. 3.

The primary bile acids increased CXCL16 expression produced by liver sinusoidal endothelial cells. Increased CXCL16 leads to activated CXCR6+ NKT cells, which have anti-tumor activity via IFN-γ production.77 Abbreviations: NKT, natural killer T; IFN, interferon.