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. 2020 Nov 27;11(1):1833644. doi: 10.1080/20008198.2020.1833644

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© 2020 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Schematic model of multilevel interactions between the human stress and circadian system

3β-HSD: 3β-Hydroxysteroid dehydrogenase; ANS: autonomic nervous system; ACTH: adrenocorticotropic hormone; APG: anterior pituitary gland; AVP: arginine vasopressin; CA: catecholamines; CAN: central autonomic network; CRH: corticotropin releasing hormone; HPA: hypothalamus-pituitary-adrenal axis; HT: hypothalamus; GC: glucocorticoids; GR: glucocorticoid receptor; MLT: melatonin; MT: MLT receptor; MR: mineralocorticoid receptor; PGL: pineal gland; PVN: paraventricular nucleus; RHT: retinohypothalamic tract; RNcl: raphe nucleus; SCG: superior cervical ganglia; SCN: suprachiasmatic nucleus.