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. 2020 Aug 25;25(1):54–61. doi: 10.29252/ibj.25.1.54

Table 3.

Analysis of the association between ABCB1 SNPs and CML risk

SNP/model Genotype Controls
n = 100 (%)
Patients
n = 98 (%)
p *
value
OR *
(95% CI)
c.1236C>T Codominant TT 29 (29.0) 25 (25.5) Ref 1
CT 53(53.0) 54 (55.1) 0.88 1.08 (0.53-2.20)
CC 18(18.0) 19(19.4) 0.88 (0.35-2.21)
Dominant TT 29 (29.0) 25 (25.5) Ref 1
CT/CC 71 (71.0) 73 (74.5) 0.93 1.03 (0.52-2.03)
Recessive TT/CC 82 (82.0) 79 (80.6) Ref 1
CT 18 (18.0) 19 (19.4) 0.65 0.83 (0.38-1.84)
Alleles T 111 (51.6) 104 (48.4) Ref 1
C 89 (49.2) 92 (50.8) 0.85 0.96 (0.62-1.476)
c.3435C>T Codominant TT 29 (29.0) 22 (22.4) Ref 1
CT 49 (49.0) 57 (58.2) 0.66 1.13 (0.54-2.33)
CC 22 (22.0) 19 (19.4) 0.78 (0.31-1.94)
Dominant TT 29 (29.0) 22 (22.4) vef 1
CT/CC 71 (71.0) 76 (77.5) 0.95 1.02 (0.51-2.05)
Recessive TT/CT 78 (78.0) 79 (80.6) Ref 1
CC 22 (22.0) 19 (19.4) 0.4 0.72 (0.34-1.55)
Alleles T 107 (51.4) 101 (48.6) Ref 1
C 93 (49.5) 95 (50.5) 0.66 0.9 (0.59-104)

Ref, reference; *logistic regression model adjusted for sex, age, and smoking status