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. 2020 Dec 10;23(2):130. doi: 10.3892/mmr.2020.11769

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Copyright: © He et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — miR-182-5p reverses the effects of BNIP3 on radioresistance of nasopharyngeal carcinoma cells. (A) Cell viability was measured in the transfected 5-8F and 5-8F-R cells following IR treatment at the indicated doses. (B) Wound healing assays were performed to assess the migratory ability of the 5-8F and 5-8F-R cells treated with 4 Gy IR. *P<0.05, **P<0.001 vs. NC; ^##P<0.001 vs. si-BNIP3 group in 5-8F cells; ^$$P<0.001 vs. BNIP3 OE group in 5-8F-R cells. BNIP3, BCL2/adenovirus E1B 19 kDa protein-interacting protein 3; NC, negative control; miRNA/miR, microRNA; si-, small interfering RNA; OE, overexpression; IR, irradiation.