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. 2020 Dec 10;7:604766. doi: 10.3389/fmolb.2020.604766

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Copyright © 2020 Bian, Ni, Zhu, Song, Zhang, Ni and Zheng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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YTHDF1 promoted epithelial–mesenchymal transition (EMT) and AKT/glycogen synthase kinase (GSK)-3β/β-catenin signaling of hepatocellular carcinoma (HCC) cells. (A–C) Gene set enrichment analysis (GSEA) was conducted to predict the potential functions and pathways regulated by YTHDF1 based on The Cancer Genome Atlas (TCGA) datasets. (D) The EMT markers were detected by Western blotting. (E) The markers of AKT/GSK-3β and Wnt/ β-catenin were detected by Western blotting.