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. 2020 Nov 26;12(12):3638. doi: 10.3390/nu12123638

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Effect of Siraitia grosvenorii residual extract (NHGR) on the protein expression of filaggrin and chemokine and activation of ERK, JNK, and p38 MAPKs from TNF-α/IFN-γ-induced HaCaT cells. (a) Cell viability, (b) filaggrin expression, (c) chemokine (RANTES, TARC, and TSLP) production, and (d) phosphorylation of ERK, JNK, and p38 MAPKs. Expression of protein and phosphorylation of MAPKs were examined. Results are showed as means ± SEM (n = 3). ^# p < 0.05, ^## p < 0.01 and ^### p < 0.001 versus vehicle; * p < 0.05, ** p < 0.01 and *** p < 0.001 versus TNF-α/TNF-γ-treated control.

Effect of Siraitia grosvenorii residual extract (NHGR) on the protein expression of filaggrin and chemokine and activation of ERK, JNK, and p38 MAPKs from TNF-α/IFN-γ-induced HaCaT cells. (a) Cell viability, (b) filaggrin expression, (c) chemokine (RANTES, TARC, and TSLP) production, and (d) phosphorylation of ERK, JNK, and p38 MAPKs. Expression of protein and phosphorylation of MAPKs were examined. Results are showed as means ± SEM (n = 3). ^# p < 0.05, ^## p < 0.01 and ^### p < 0.001 versus vehicle; * p < 0.05, ** p < 0.01 and *** p < 0.001 versus TNF-α/TNF-γ-treated control.