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Possible S-nitrosation site on TIR1 not consistent with phenotype of GSNOR 1–3 mutant [1,44]. (A) shows S-nitrosylation sites on TIR1 thought to be capable of upregulating auxin production hypothesized to increase growth however (B) shows that in application the GSNOR knock out leads to stunted growth, evidence in favor of GSNORs pleiotropic effects.
