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. 2020 Nov 27;9(12):1187. doi: 10.3390/antiox9121187

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Changes in apoptosis in WT and NLRP3^−/− mice during aging and melatonin treatment. (A) Protein levels of p53. (B) Protein levels of caspase 9. (C) Protein levels of Bax. (D) Protein levels of Bcl2. (E) Bax/Bcl2 ratio. Experiments were performed in hearts of young (Y), early-aged (EA), early-aged with melatonin (EA + aMT), old-aged (OA), and old-aged with melatonin (OA + aMT) wild type and NLRP3^−/− mice. Data are expressed as means ± SEM (n = 7 animals/group). * p < 0.05, ** p < 0.01 vs. Y; ^# p < 0.05, ^## p < 0.01 vs. group without melatonin treatment.