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. 2020 Nov 24;9(12):1173. doi: 10.3390/antiox9121173

Figure 1.

Figure 1

Effects of Hawthorn (HAW) on CCl4-induced liver damage in rats. (A) representative images of hematoxylin and eosin (H & E)-stained hepatic sections of the different groups (100×), Scale bar equals 100 µm. Histopathological evidence of CCl4-induced liver damage in rats showing area of necrosis with inflammatory cell infiltration (arrow), and diffuse fatty change (star), (B) severity score of necrosis, (C) inflammation, and (D) fatty metamorphosis. (E) liver weight, (F) body weight, and (G) liver/body weight ratio. Serum markers of liver damage aspartate aminotransferase (AST) (H), alanine aminotransferase (ALT) (I), gamma-glutamyl transferase (GGT) (J) activities and total bilirubin (K), and albumin (L) levels. Data are presented as means ± SEM. (n = 6 per group). a is p < 0.05 vs. control group, b is p < 0.05 vs. CCl4 group, and c is p < 0.05 vs. HAW group.