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Process of Aβ aggregation and β-amyloid plaque formation. The Aβ peptide, once excised from APP, is prone to misfolding and self-aggregation. Misfolded Aβ monomers aggregate into small soluble oligomers. The oligomers interact to form protofibrils, which grow to form mature fibrils. Eventually the fibrils aggregate, forming the β-amyloid plaques.
