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. 2020 Dec 23;4(2):e202000804. doi: 10.26508/lsa.202000804

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© 2020 Dunbar et al.

This article is available under a Creative Commons License (Attribution 4.0 International, as described at https://creativecommons.org/licenses/by/4.0/).

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Figure S6. — (A) Extracts from MV4.11 and DLD-1 cells treated with IMiDs (10 μM for 24 h) were resolved by SDS–PAGE and subjected to Western blotting with the indicated antibodies. (B) Densitometry of CK1α protein abundance in MV4.11 cells treated with IMiDs (10 μM for 24 h). CK1α protein abundance was normalised to GAPDH protein abundance and represented as a fold change compared to untreated cells. Data presented as scatter graph illustrating individual data points with an overlay of the mean ± SD. Statistical analysis was performed using a Student’s unpaired t test and by comparing fold change between untreated cells and cells treated with IMiDs (10 μM for 24 h). Statistically significant P-values are denoted by asterisks (* < 0.05, ** < 0.01, *** < 0.001, **** < 0.0001).

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