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. 2020 Dec 23;14:1753466620962665. doi: 10.1177/1753466620962665

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© The Author(s), 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 6. — The treatments with the Rho-kinase and anti-IL17 attenuates the expression of remodeling markers in vessel in chronic allergic pulmonary inflammation induced by ovalbumin in mice. In OVA group there was an increase compared with the saline group in the expression of MMP-12 and TIMP-1. The data are presented as dot plots with standard deviations. Representative photomicrographs of MMP-12, TIMP-1 and TGF-β for each group. The red arrows indicate the marked area in immunohistochemistry.

IL, interleukin; MMP, metalloproteinase; TIMP, metalloproteinase inhibitor; TGF-β, transforming growth factor; OVA, animals induced to chronic allergic inflammation by ovalbumin; OVA-anti-IL17, animals induced to chronic allergic pulmonary inflammation and use of anti-IL17; OVA-RHO inhibitor, animals induced to chronic allergic pulmonary inflammation and use of Rho-kinase inhibitor; OVA-RHO inhibitor-anti-IL17, animals induced to chronic allergic pulmonary inflammation and use of Rho-kinase inhibitor and anti-IL17; SAL, saline group.