Skip to main content
. 2020 Sep 2;13(6):952–968. doi: 10.1093/ckj/sfaa157

Table 1.

Knowledge gaps

  1. Is potassium-induced natriuresis (with NCC dephosphorylation) preserved in CKD?

  2. Does CKD cause a switch from urinary K+ excretion to gastro-intestinal K+ excretion or intracellular redistribution?

  3. In CKD, do the beneficial effects of dietary K+ outweigh the risks of hyperkalaemia?

  4. Are the positive effects of dietary K+ mediated by plasma K+ and, if so, are they concentration dependent?

  5. What is the effect of dietary K+ on phosphate balance and fibroblast growth factor 23?