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. 2020 Dec 29;21(Suppl 11):793. doi: 10.1186/s12864-020-07207-4

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Examination of RUNX1T1-RUNX1 fusion in Nanopore long-read RNA-Seq data and Sanger sequencing data on a patient with AML. a The IGV plot for the gene fusion at the genomic region around RUNX1T1. b The IGV plot for the gene fusion at the genomic region around RUNX1. c the result from Sanger sequencing. The vertical dotted line indicates the genomic location where breakpoint occurs. The Sanger sequence in (c) is the complementary of the sequences in (a) and (b)