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editorial

. 2020 Dec 28;6(3):e001454. doi: 10.1136/rmdopen-2020-001454

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© Author(s) (or their employer(s)) 2020. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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Proposed mechanism whereby 1,25(OH) D -VDR (D-hormone) signalling acts on the respiratory system during the COVID-19 infection. 1,25(OH) D, 1,25-hydroxyvitamin D; ARTI, acute respiratory tract infection; IFN-gamma, inteferon gamma; IL-6, inteleukin 6; IL10, interleukin; IL -12, interleukin 12; IL-17, interleukin 17; Treg, T regulatory cells; TNF, tumour necrosis factor; UV, ultraviolet; VDR, vitamin D receptor.