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. 2019 Dec 13;71(11):2858–2868. doi: 10.1093/cid/ciz1186

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© The Author(s) 2019. Published by Oxford University Press for the Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 7. — Microbial taxa, functional pathway, and resistance gene β-diversity are lower in cotrimoxazole-treated human immunodeficiency virus–exposed, uninfected (HEU) infants (CTX-T) than in HEU infants not treated with cotrimoxazole (CTX-N). Data are shown for β-diversity calculated from microbial taxonomic profiles, functional metabolic pathways, and resistance genes. Distributions show the difference at each collection timepoint between the CTX-T cohort’s Bray-Curtis dissimilarities and the CTX-N cohort’s Bray-Curtis dissimilarities for 5000 bootstrapped subsamples. The black points are the mean value for this distribution, and black lines are 95% confidence intervals.