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. 2021 Jan 4;48(6):1890–1901. doi: 10.1007/s00259-020-05154-6

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© The Author(s), under exclusive licence to Springer-Verlag GmbH, DE part of Springer Nature 2021

This article is made available via the PMC Open Access Subset for unrestricted research re-use and secondary analysis in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the World Health Organization (WHO) declaration of COVID-19 as a global pandemic.

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Fig. 4 — Results of the correlation analyses performed between the cerebral glucose metabolism and the severity of olfactory dysfunction and between the cerebral glucose metabolism and the duration of anosmia. (a) Regression plots of the severity of olfactory dysfunction assessed by the identification test score (Top) and adjusted metabolic responses obtained by considering the peak voxel in the right FEF ([30 6 48], Pearson’s correlation: r = 0.97, p < .001) and in the left orbitofrontal cortex ([− 12 68 0], Pearson’s correlation: r = 0.94, p < .001). (b) Regression plots of the duration of anosmia (Bottom) and adjusted metabolic responses obtained by considering the peak voxel in the right DLPFC ([38 32 34], Pearson’s correlation: r = 0.94, p < .001) and in the right orbitofrontal cortex ([34 32 − 10], Pearson’s correlation: r = 0.9, p < .001)