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. 2020 May 10;45(13):2248–2256. doi: 10.1038/s41386-020-0693-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a The block-wise beta of Faces>Shapes within the ROIs of controls and individuals with autism spectrum disorder (ASD) for the two drug conditions (placebo and citalopram) were extracted and here the means are plotted with standard error bars. b Using a regression approach to summarise the decrease in activations across time (i.e., habituation), absolute habituation indices were obtained and significant group × drug interaction effects on absolute habituation indices could be observed in all ROIs, including basolateral amygdala (BLA), centromedial amygdala (CMA), superficial amygdala (SFA), nucleus accumbens (NAc), subgenual anterior cingulate cortex (sgACC), ventral ACC (vACC), rostral ACC (rACC) and anterior ventromedial prefrontal cortex (avmPFC) (p corrected < 0.001). Significance for post-hoc analyses corrected for multiple comparisons is demonstrated with asterisks. *p < 0.05; ***p < 0.001.