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. 2021 Jan 6;13:3. doi: 10.1186/s13148-020-00996-1

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Identification of ChIP-seq differential binding genes, GO annotation, and pathway analysis. a Venn diagram indicating the overlap from MACS and SICER between control and DCM samples, respectively. b-d Biological process GO terms of differential target genes. b LMNA differential target genes in normal tissues. c Differential target genes of LAP2α-lamin A/C complexes in DCM. d Differential target genes of LAP2α-lamin A/C complexes in control samples. e–g Pathway analysis of differential target genes. The color of the circle represents the p value, and the size of the circle refers to the number of genes enriched in the same entry. e LMNA differential target genes in normal tissues. f Differential target genes of LAP2α-lamin A/C complexes in DCM. g Differential target genes of LAP2α-lamin A/C complexes in control samples